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IL-25 treatment improves metabolic syndrome

IL-25 treatment improves metabolic syndrome in high fat diet and genetic models of obesity- read for more information ....

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A biomarker-authenticated model of schizophrenia implicating NPTX2 loss of function

A biomarker-authenticated model of schizophrenia implicating NPTX2 loss of function
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New genomic research facility to support food security efforts in UAE and abroad

New genomic research facility to support food security efforts in UAE and abroad
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Underlying mechanisms of anlotinib resistance in NSCLC

Underlying mechanisms of anlotinib resistance in NSCLC
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Pooled CRISPR screening identifies m6A as a positive regulator of macrophage activation

Cd86), were down-regulated in
Mettl3-deficient cells (Fig. 1G), suggesting that METTL3 has a critical function in controlling the innate immune response of Raw 264.7 macrophages.
To further confirm the biological role of the m
6A modification in macrophages,
Mettl3 conditional KO (CKO) mice were generated by crossing
Mettl3
flox/flox mice with mice expressing Cre recombinase under the control of lysozyme 2 promoter (
Lyzm-Cre). We have documented the loss of both the METTL3 protein and the overall m
6A modification in bone marrow–derived macrophages (BMDMs) from
Mettl3
flox/flox;
Lyzm-Cre mice (fig. S1I). No differences in the frequency of major immune cell populations were observed between ....

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