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Hub candidate network for hepatocellular carcinoma

Hub candidate network for hepatocellular carcinoma
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Yuhan Chen , Chen Research Team , Clinical Proteomic Tumor Analysis Consortium , Research Output , International Cancer Genome Consortium , Southern Medical University , Cancer Genome Atlas , Gene Expression Omnibus , சென் ஆராய்ச்சி அணி , ஆராய்ச்சி ஔட்‌புட் , சர்வதேச புற்றுநோய் மரபணு கூட்டமைப்பு , தெற்கு மருத்துவ பல்கலைக்கழகம் , புற்றுநோய் மரபணு அட்லஸ் , கீந் வெளிப்பாடு சர்வபுலம் ,

Study shows how m6A regulators influence metabolic heterogeneity of hepatocellular carcinoma

Study shows how m6A regulators influence metabolic heterogeneity of hepatocellular carcinoma
Cancer cells acquire growth advantages over normal cells in myriad ways. Changes in cell programming allow these cells to grow in an uncontrolled fashion, thereby forming the cancer mass.
Hepatocellular carcinoma (HCC), being highly malignant and invasive with a high recurrence rate and drug-resistant phenotype, is one of the most dreadful cancers. Understanding the underlying molecular mechanisms is crucial to design therapeutic interventions and to predict patient prognosis.
Cancer cells use metabolic, immunogenic, or growth-related strategies, which can be controlled by a number of alterations in the cell process. Of these, post-transcriptional RNA modification has recently sparked interest among cancer biologists. N6-methyladenosine (m6A) methylation, a type of RNA modification, remains the most abundant epigenetic (non-DNA related) modification in eukaryotic cells. ....

Lun Xiu Qin , Emily Henderson , Fudan University , International Cancer Genome Consortium , Cancer Biology , Cancer Genome Atlas , Hepatocellular Carcinoma , Liver Cancer , லூந் க்ஷிு குயின் , எமிலி ஹென்டர்சன் , சர்வதேச புற்றுநோய் மரபணு கூட்டமைப்பு , புற்றுநோய் உயிரியல் , புற்றுநோய் மரபணு அட்லஸ் , வளர்சிதை மாற்றம் , கல்லீரல் புற்றுநோய் , அறுவை சிகிச்சை ,

Cancer control: Non-DNA changes induce metabolism variations in hepatocellular carcinomas

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Credit: Cancer Biology & Medicine
Cancer cells acquire growth advantages over normal cells in myriad ways. Changes in cell programming allow these cells to grow in an uncontrolled fashion, thereby forming the cancer mass. Hepatocellular carcinoma (HCC), being highly malignant and invasive with a high recurrence rate and drug resistant phenotype, is one of the most dreadful cancers. Understanding the underlying molecular mechanisms is crucial to design therapeutic interventions and to predict patient prognosis.
Cancer cells use metabolic, immunogenic, or growth-related strategies, which can be controlled by a number of alterations in the cell process. Of these, post-transcriptional RNA modification has recently sparked interest among cancer biologists. N6-methyladenosine (m6A) methylation, a type of RNA modification, remains the most abundant epigenetic (non-DNA related) modification in eukaryotic cells. Abnormal activity of proteins that handle m6A re ....

Wei Qin , Lun Xiu Qin , Ryan Fu , Qiongzhu Dong , Lunxiu Qin , Jing Xu , Xiaotian Shen , Institutes Of Biomedical Sciences , Fudan University , International Cancer Genome Consortium , Shanghai Medical College , Huashan Hospital Cancer Metastasis Institute , Cancer Biology , Cancer Genome Atlas , Shun Wang , General Surgery , Huashan Hospital , Cancer Metastasis Institute , Biomedical Sciences , Cell Biology , Developmental Reproductive Biology , Medicine Health , வெய் குயின் , லூந் க்ஷிு குயின் , யான் ஃபூ , ஜிங் ஐயூ ,