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Stroke of Luck: Therapeutic Target Discovered in Blood–Brain Barrier


 
Scientists from Japan and the United States have identified a new mechanism of blood-brain barrier degradation in the post-stroke brain, involving acrolein-induced modifications of proheparanase. This discovery could lead to the production of newer and more effective drugs for stroke-related disorders. Credit: Toubibe from Pixabay
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The blood-brain barrier prevents immune cells from circulating freely in the brain, and the breakdown of its function is a major cause of post-stroke inflammation. Now, for the first time, researchers have identified how a toxic stroke byproduct, acrolein, could activate the barrier-degrading enzyme proheparanase (proHPSE). The research group has discovered that proHPSE degrades the glycocalyx within the post-stroke brain s blood vessels, providing hope for new and effective therapeutics against post-stroke inflammation. ....

United States , Kazuei Igarashi , Tomomi Furihata , Takehiro Suzuki , Toshihiko Toida , Kyohei Higashi , Naoshi Dohmae , Robertj Linhardt , Tokyo University Of Pharmacy , Amine Pharma Research Institute , Rensselaer Polytechnic Institute , Life Sciences , Tokyo University Of Science , Chiba University , Associate Professor Kyohei Higashi , Tokyo University , Biological Chemistry , Sustainable Resource Science , ஒன்றுபட்டது மாநிலங்களில் , டோக்கியோ பல்கலைக்கழகம் ஆஃப் மருந்தகம் , அமீன் பார்மா ஆராய்ச்சி நிறுவனம் , ரென்சீலர் பாலிடெக்நிக் நிறுவனம் , வாழ்க்கை அறிவியல் , டோக்கியோ பல்கலைக்கழகம் ஆஃப் அறிவியல் , சிபா பல்கலைக்கழகம் , டோக்கியோ பல்கலைக்கழகம் ,