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A noncanonical IRAK4-IRAK1 pathway counters DNA damage–induced apoptosis independently of TLR/IL-1R signaling

A noncanonical IRAK4-IRAK1 pathway counters DNA damage–induced apoptosis independently of TLR/IL-1R signaling
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Mutation in Hailey-Hailey Disease

Hepatocellular carcinoma in growth hormone receptor KO mouse

[Full text] miR-744-5p Inhibits Multiple Myeloma Proliferation, Epithelial Mesench

1 2Medical Records Management Division, Chongqing University Cancer Hospital, Chongqing, People’s Republic of China; 3Intensive Care Unit, Chongqing University Cancer Hospital, Chongqing, People’s Republic of China; 4Pharmacy Services, Chongqing University Cancer Hospital, Chongqing, People’s Republic of China These authors contributed equally to this work Correspondence: Dehong Huang Department of Hematology and Oncology, Chongqing University Cancer Hospital, No. 181 Hanyu Road, Chongqing, People’s Republic of China Tel +86 23-65311341 Purpose: This study investigated the function and molecular mechanisms of miR-744-5p in multiple myeloma (MM). Methods: miR-744-5p and SRY-related high-mobility-group box 12 (SOX12) expression in clinical tissues and MM cells was monitored by quantitative real-time polymerase chain reactions and Western blot. miR-744-5p expression in MM cells was regulated by transfection. Cell proliferation was researched by cell counting kit-8 as

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