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Cancer Drug Resistance Explained by Research

Cancer Drug Resistance Explained by Research
miragenews.com - get the latest breaking news, showbiz & celebrity photos, sport news & rumours, viral videos and top stories from miragenews.com Daily Mail and Mail on Sunday newspapers.

Nate traasethWilliam marsigliaYingkai zhangMoosa mohammadiAlida beschNational institutes of healthNational science foundationUniversity of alabama at birminghamDepartment of chemistrySimons centerProceedings of the national academy sciencesTraaseth labYork universityOujiang laboratory in wenzhouNew york universityNational academy

NYU Launches Simons Center for Computational Physical Chemistry

NYU Launches Simons Center for Computational Physical Chemistry
miragenews.com - get the latest breaking news, showbiz & celebrity photos, sport news & rumours, viral videos and top stories from miragenews.com Daily Mail and Mail on Sunday newspapers.

United statesYingkai zhangStefano martinianiTamar schlickMark tuckermanGlen hockyZlatko baSimons centerDepartment of chemistryCourant institute of mathematical sciencesSimons foundationUniversity of calabriaUniversity of chicagoYork universityComputational physical chemistryCourant institute

Prototype Shows Promise As Approach to Countering Pancreatic Cancer

Prototype Shows Promise As Approach to Countering Pancreatic Cancer News provided by Share this article Share this article NEW YORK, May 11, 2021 /PRNewswire/ A research team has designed a molecule with potential to interfere in a new way with altered proteins that cause abnormal growth in 35 percent of pancreatic cancers. Published online in Nature Communications on May 11, a new study found that a molecule called a monobody clings to cancer-causing versions of the KRAS protein and keeps them from transmitting their signals. Changes in the DNA of the KRAS gene – which encodes a molecular switch that toggles between active and inactive states to regulate growth – cause the related protein to become stuck in the on mode. Cells with such mutations continually multiply and give rise to cancer.

United statesCarmine fedeleSteven tsaiYingkai zhangGregory williamsKai wen tengJohn obryanAkiko koideShohei koideBenjamin neelXuben houTakamitsu hattoriNational institutes of healthLangone health perlmutter cancer centerDepartment of biochemistryDepartment of cell

Antioxidant Found in Green Tea May Increase Levels of p53

Read Time: An antioxidant found in green tea may increase levels of p53, a natural anti-cancer protein, known as the âguardian of the genomeâ for its ability to repair DNA damage or destroy cancerous cells. Published today in Nature Communications, a study of the direct interaction between p53 and the green tea compound, epigallocatechin gallate (EGCG), points to a new target for cancer drug discovery. âBoth p53 and EGCG molecules are extremely interesting. Mutations in p53 are found in over 50% of human cancer, while EGCG is the major anti-oxidant in green tea, a popular beverage worldwide,â said Chunyu Wang, corresponding author and a professor of biological sciences at Rensselaer Polytechnic Institute. âNow we find that there is a previously unknown, direct interaction between the two, which points to a new path for developing anti-cancer drugs. Our work helps to explain how EGCG is able to boost p53âs anti-cancer activity, opening the door to dev

Jing zhaoUnited statesUniversity of massachusettsLauren gandyYingkai zhangCurt brenemanJianhan chenChunyu wangWeihua jinSozanne solmazXinyue liuStewart lohMichael cosgroveYuanyuan xiaoAlan blaneyLufeng yan

Green Tea Compound Aids Tumor-Suppressing, DNA-Repairing Protein

Research offers new lead for cancer drug discovery An antioxidant found in green tea may increase levels of p53, a.

Jing zhaoUnited statesUniversity of massachusettsLauren gandyYingkai zhangCurt brenemanJianhan chenChunyu wangWeihua jinSozanne solmazXinyue liuStewart lohMichael cosgroveYuanyuan xiaoAlan blaneyLufeng yan