Recent studies by Zampaloni et al. and Pahil et al. published in the journal Nature describe a novel method of inhibiting the growth of Gram-negative bacteria such as Acinetobacter using antibiotics consisting of macrocyclic peptides that target the bacterial protein bridge machinery that transports lipopolysaccharides from the cytoplasm to the outer membrane.
i mean nanograms, which is containing many biological products are able to enter our nuclear s, integrate itself into dna and cause things like cancer and autoimmune diseases, that s impossible and here s why. if you think about a cell as like a sunny side up egg and the white part is the cytoplasm and the nucleus is the yoke, if these fragmented dm dnas get into the cytoplasm they would be instantly eliminated because our cytoplasm hates dna, even if it got that, enter the nuclear, which is impossible because it doesn t have a nuclear accessible and gets into the nuclear s, it would have to integrate into the dna, none of that is contained in the fragments and let s also
Gastric cancer, a significant global health burden, claims over 7% of cancer-related deaths annually. Although only 1-3% of cases have a genetic basis, understanding these genetic drivers is crucial for developing preventative strategies.
Back in 2018, the lab of Christine Mayr, MD, PhD, at Memorial Sloan Kettering Cancer Center (MSK) introduced the world to a key cellular component that had been hiding in plain sight.
A surprising mechanism that makes some cancers treatment-resistant has been discovered by Weill Cornell Medicine and NewYork-Presbyterian investigators.