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Restoring cholesterol levels could be a new therapeutic approach for diseases linked to TDP-43 protein Researchers in Singapore have discovered that brain cells cannot maintain the cholesterol-rich myelin sheath that protects and insulates neurons in the absence of a protein called TDP-43. The study, which will be published August 4 in the Journal of Cell Biology ( JCB), suggests that restoring cholesterol levels could be a new therapeutic approach for diseases associated with TDP-43. The TDP-43 protein is linked to multiple neurodegenerative diseases, including amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). TDP-43 plays many vital roles within cells, but, under certain circumstances, it can clump together to form toxic aggregates that damage cells and prevent TDP-43 from performing its normal functions. TDP-43 aggregates are found in the brains of most ALS patients and ~45% of FTD patients and are also linked to several other neurodegenerative disorders,

National university of singaporeSingapore generalEmily hendersonYong loo lin school of medicineCell biologyChien lingYong loo lin schoolNational universityதேசிய பல்கலைக்கழகம் ஆஃப் சிங்கப்பூர்எமிலி ஹென்டர்சன்யோங் லூ லின் பள்ளி ஆஃப் மருந்துசெல் உயிரியல்யோங் லூ லின் பள்ளிதேசிய பல்கலைக்கழகம்