The Japanese liberal political magazine Sekai announced that it is being revamped for the first time in a quarter of a century to achieve greater gender parity and reach new audiences.
Scientists decipher missing pieces of the reward system puzzle to increase motivation
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Novel technique decodes mechanisms controlling executive functions of the primate brain
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Shahriar SheikhBahaei, Ph.D.
Dr. SheikhBahaei’s interest in neuroscience stemmed from the usual combination of an aptitude for science and a medical problem (stuttering) that brought him into bioscience at a young age. Dr. SheikhBahaei received his bachelor’s degree from the University of California at Berkeley, where he worked with Dr. Bob Zucker on the regulation of neurotransmitter release and Dr. John Rubenstein (UC San Francisco) on development of GABAergic neurons in basal ganglia. Dr. SheikhBahaei completed his doctoral studies in Neuroscience (2017) jointly under NIMH/NINDS – University College London (UCL) Graduate Partnership Program where he worked with Drs. Jeffrey Smith (NINDS) and Alexander Gourine (UCL). His graduate studies were on how astrocytic networks control activities of respiratory motor circuits within the brainstem. After short postdoctoral research at NINDS, Dr. SheikhBahaei became an Independent Research Scholar in 2019. In collaboration with the lab
Amyloid protein (orange) forms clumps among neurons (blue). Amyloid in the brain is one of the proteins associated with Alzheimer’s disease, but amyloid clumps in other parts of the body can cause other illnesses. © Juan Gärtner/Adobestock.com
A small, light-activated molecule recently tested in mice represents a new approach to eliminating clumps of amyloid protein found in the brains of Alzheimer’s disease patients. If perfected in humans, the technique could be used as an alternative approach to immunotherapy and used to treat other diseases caused by similar amyloids.
Researchers injected the molecule directly into the brains of live mice with Alzheimer’s disease and then used a specialized probe to shine light into their brains for 30 minutes each day for one week. Chemical analysis of the mouse brain tissue showed that the treatment significantly reduced amyloid protein. Results from additional experiments using human brain samples donated by Alzheimer’s disease pat