The research paper explores the multifaceted role of the IL-17 family in immune response, covering everything from infection control to pathological conditions like autoimmune diseases and cancer. Future therapies may exploit IL-17's unique signaling pathways to offer more targeted and cost-effective treatments.
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Coronavirus affects people differently – some infected develop life-threatening disease, while others remain asymptomatic. And a year aftere COVID-19 emerged, it’s still unclear why.
To try and answer this question, researchers have started looking at the genetics of people who get COVID-19, and identifying links between developing the disease and variations in specific parts of our DNA. This raises the possibility that some of what makes people susceptible to COVID-19 lies in their genes.
This wouldn’t be surprising. Genetic variation plays a role in susceptibility to a number of diseases, from HIV to malaria to TB. Researchers know this because they hunt for variations of interest by comparing people’s entire DNA sequences – their genomes – to see whether certain variations coincide with certain disease outcomes. Such analyses are called genome-wide association studies.