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I have no idea. you re going up to have an infusion at the clinic. oh, thank you. that s exciting. come on, rodney, let s get going. mavis is one of nearly 2,000 patients who took part in a pivotal trial of an experimental alzheimer s treatment. so, i m just going to show- you the hippocampus, and that s where all our short term memory is stored. - and what you can see is that structure is very small - and shrunken and replaced by fluid because of- the disease. alzheimer s gradually destroys key areas of the brain involved in memory and understanding. the damage is driven by the build up of a rogue protein called amyloid, which clumps around neurons in the brain. the new drug lecanemab binds to amyloid, which prompts immune cells to attack and clear it from the brain. access to the drug could be severely limited, in part because specialist tests required to screen patients for amyloid ....
Because of the disease. alzheimer s gradually destroys key areas of the brain involved in memory and understanding. david is one of nearly 2,000 patients who took part in a major trial of a new drug, lecanemab. as normal, if you just lift your arm up there for me. it involved having an infusion every two weeks. lecanemab didn t stop alzheimer s. i lljust get you started here. ..but over 18 months, it slowed its progression by about a quarter. this is so exciting because now we re getting results, the first results that are indicating that the drug is successfully treating the underlying cause and is slowing down the symptoms of cognitive impairment, and also the behavioural symptoms associated with alzheimer s disease. in alzheimer s, progressive damage is inflicted on the brain by the build up of a rogue protein called amyloid. ....