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SARS-CoV-2 spike protein could be speeding up Alzheimer s and other brain diseases, says new study

Swedish researchers find that SARS-CoV-2's spike protein amyloids can accelerate the formation of harmful protein aggregates related to neurodegenerative diseases like Creutzfeldt-Jakob and Alzheimer's. The study suggests a potential link between COVID-19 and increased cases of these brain disorders.

How SARS-CoV-2 hijacks Fragile X proteins to fuel infection: New clues in COVID-19 and genetic disorders

Study explores the interaction between SARS-CoV-2's non-structural protein 3 (NSP3) and proteins related to Fragile X mental retardation (FMRPs), shedding light on COVID-19 pathophysiology. The research suggests that the virus exploits these host cell proteins for effective infection and possibly interferes with antiviral defense mechanisms, offering new insights into the genetic basis of Fragile X syndrome.

SARS-CoV-2 Eris variant spreads faster and dodges immunity

Study investigates the characteristics of the EG.5.1 "Eris" variant of SARS-CoV-2, finding it more transmissible among hamsters and slightly better at evading immunity compared to the XBB.1.5 variant. The results suggest that these properties could be driving the variant's increasing prevalence in human populations.

Can ChatGPT be a diabetes consultant? Study probes the potential and pitfalls

This comprehensive study explores ChatGPT's ability to answer questions about diabetes, comparing it to human experts in a Danish diabetes center. While familiar users could more often spot the AI-generated answers, the research underscores the need for rigorous evaluation and safe integration of AI in healthcare.

Study unveils previously unknown urinary biomarkers, revolutionizing early detection and treatment of viral infections like COVID-19

This recent study identifies new urinary biomarkers for detecting acute COVID-19 using spectroscopic analysis. The findings reveal that these biomarkers could offer valuable tools for early diagnosis, understanding the severity of the disease, and possibly contribute to endogenous immunological defense mechanisms against SARS-CoV-2.

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