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What really killed COVID-19 patients: it wasn

By applying machine learning to medical record data, scientists found that secondary bacterial pneumonia that does not resolve was a key driver of death in patients with COVID-19. It may even exceed death rates from the viral infection itself. The scientists also found evidence that COVID-19 does not cause a “cytokine storm,” so often believed to cause death.

Why COVID-19 pneumonia lasts longer, causes more damage than typical pneumonia

Scientists identify target to treat COVID pneumonia and reduce severity Clinical trials with new experimental drug to begin early in 2021 Goal is to develop treatments that make COVID-19 no worse than a common cold First comparison between immune mechanisms driving COVID-19 pneumonia with other pneumonias CHICAGO - Bacteria or viruses like influenza that cause pneumonia can spread across large regions of the lung over the course of hours. In the modern intensive care unit, these bacteria or viruses are usually controlled either by antibiotics or by the body s immune system within the first few days of the illness. But in a study published in

COVID-19 pneumonia causes more damage than typical pneumonia, says study

COVID-19 pneumonia causes more damage than typical pneumonia, says study ANI | Updated: Jan 12, 2021 23:19 IST Washington [US], January 12 (ANI): Researchers from the Northwestern University claims that COVID-19pneumonia spreads like multiple wildfires, leaving destroyed lung tissue in its wake. COVID pneumonia is significantly different from pneumonia caused by other causes, reports a new study. The infection leaves damage in its wake and fuels the fever, low blood pressure and damage to the kidneys, brain, heart and other organs in patients with COVID-19. Scientists discovered a target for treating COVID pneumonia. Bacteria or viruses like influenza that cause pneumonia can spread across large regions of the lung over the course of hours. In the modern intensive care unit, these bacteria or viruses are usually controlled either by antibiotics or by the body s immune system within the first few days of the illness.

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