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Researchers at the University of Malta have found that depletion of ACE2 receptors, which permit SARS-CoV-2 to enter cells and cause COVID-19, is behind the neuromuscular complications experienced by a significant number of COVID-19 patients, according to a study published in the journal Biochimica et Biophysica Acta. ACE2 depletion is thought to be linked to lasting effects of the disease, including fatigue, breathlessness, brain fog and muscle weakness.
New research paves the way for new strategies to address long-term COVID-19 complications. Researchers from the University of Malta have identified a possible cause for the prolonged and frequently debilitating symptoms faced by individuals with long COVID-19. The recent study, which was published
Angiotensin-converting enzyme 2 (ACE2) exists in two forms: one anchored to cell membranes (mACE2) in the intestines, kidney, testis, gallbladder, and heart, and the other as a soluble variant (sACE2)
Science News: Muscular fatigue in long Covid patients has been linked to depletion of ACE2 receptors, which act as doorways for SARS-CoV-2 to infect cells and cause