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 E-Mail Clinicians and scientists have long observed that cells in overstressed hearts have high levels of the simple sugar O-GlcNAc modifying thousands of proteins within cells. Now, researchers at Johns Hopkins Medicine have found evidence in mouse experiments that these excess sugars could well be a cause, not merely a consequence or marker of heart failure. Their research found that elevated levels of O-GlcNAc made mice more prone to heart failure, but lowering levels of O-GlcNAc restored the animals risk of death and heart function to normal. Together, the investigators say, the new findings, described online in the April 27th issue of the journal

United statesNational taiwan universityT ai peiNatasha zacharaPriya umapathiC conover talbot jrJonathan grangerJohns hopkinsOlurotimi mesubiQinchuan wangElizabeth luczakNeha abrolLiliana floreaOscar reyes gaidoMark andersonChi wei