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With antibiotic-resistant bacteria on the rise, scientists have been searching for ways to shut down the Type IV secretion system (T4SS), a protein complex on the outer envelope of bacterial cells that helps them to exchange DNA with neighboring bacteria and resist antibiotics.
This seminar will highlight the alarming nature of antimicrobial drug resistance in tuberculosis and challenges in tackling different physiological states of the bacteria that cause the disease. The lecture will address some key research findings on the propsect of repurposing non-steroidal anti-inflammatory drugs (NSAIDs) to reverse extensive drug resistance in tuberculosis.