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New drug targets EMT in cancer, reducing metastasis and resistance to chemotherapy

Metastases and resistance to chemotherapy are the main causes of treatment failure and mortality in cancer patients.

Bruxelles capitaleMegan craigIevgenia pastushenkoJustine lengrandSebastiaan vanuytvenResearch instituteCancer laboratoryUniversity of lyonStem cellsCancer therapyStem cells

A new drug prevents epithelial-mesenchymal transition, metastasis and resistance to anti-cancer therapy

A new drug prevents epithelial-mesenchymal transition, metastasis and resistance to anti-cancer therapy
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Bruxelles capitaleJustine lengrandIevgenia pastushenkoPatrick mehlenSebastiaan vanuytvenUniversity of lyonCancer laboratoryResearch instituteStem cells

FAT1 mutations act as tumor suppressor and prevent cancer development, indicates study

FAT1 mutations act as tumor suppressor and prevent cancer development, indicates study Cancer metastasis, which is the dissemination of tumor cells into distant organs, is the leading cause of mortality in cancer patients. To undergo metastasis, cells must leave the primary tumor, circulate into the blood, colonize distant organs, and form distant metastasis. It has been proposed that epithelial to mesenchymal transition (EMT), a process in which epithelial cells detach from their neighboring cells, and acquire mesenchymal migrating properties, is important to initiate the metastatic cascade allowing the cancer cells to leave the primary tumor. However, the role of genetic mutations in promoting EMT is unknown.

Bruxelles capitaleCedric blanpainIevgenia pastushenkoEmily hendersonLaboratory of stem cellsStem cellsStudy first authorப்ரூக்ஸெல்ஸ் தலைநகரம்எமிலி ஹென்டர்சன்ஆய்வகம் ஆஃப் தண்டு செல்கள்தண்டு செல்கள்படிப்பு முதல் நூலாசிரியர்

Cancer: Tumor driver promoting EMT, metastasis and resistance to therapy

Cancer: Tumor driver promoting EMT, metastasis and resistance to therapy Cancer metastasis, which is the dissemination of tumor cells into distant organs, is the leading cause of mortality in cancer patients. To undergo metastasis, cells must leave the primary tumor, circulate into the blood, colonize distant organs, and form distant metastasis. It has been proposed that epithelial to mesenchymal transition (EMT), a process in which epithelial cells detach from their neighboring cells, and acquire mesenchymal migrating properties, is important to initiate the metastatic cascade allowing the cancer cells to leave the primary tumor. However, the role of genetic mutations in promoting EMT is unknown.

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