Initial extreme hyperoxia at ECMO-assisted CPR for out-of-hospital cardiac arrest predicted less favorable neurologic outcomes and survival in a registry-based analysis.
Kyushu University researchers find that one of the causes of CKD induced heart failures is the over expression of G protein-coupled receptor 68 in monocytes. Monocyte disruption was caused by the activation of the Clock gene, the key regulator of circadian rhythm via the increased level of vitamin A in the body resulting from kidney dysfunction.