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Improving Genomic Result Communication for Late-Stage Cancer

Improving Genomic Result Communication for Late-Stage Cancer
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United statesStacyw grayAmerican cancer societyDivision of clinical cancer genomicsClinical cancer genomicsMedical oncologyTherapeutics research

Why it s important to improve communication of unanticipated genomic findings to patients with late-stage cancer

Why it s important to improve communication of unanticipated genomic findings to patients with late-stage cancer
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United statesMadeline curreyStacyw grayDivision of clinical cancer genomicsIlana solomonClinical cancer genomicsMedical oncologyTherapeutics researchReturning secondary findingsLate stage cancer care

Defining Earlier Use of Immunotherapy in Patients With NSCLC

In the non–small cell lung cancer space, identifying the appropriate candidate to receive perioperative approaches, such as neoadjuvant or adjuvant therapy, remains the clinical objective.

United statesNew yorkBruna pelliniMarkg krisLeslyem heislerCharu aggarwalBalazs halmosH lee moffitt cancer centerPenn centerNy montefiore einstein cancer centerAirways malignancies researchMontefiore einstein cancer centerClinical cancer genomicsNew york lung cancers symposiumTargeted therapiesJoy ruane chair

Cisplatin/Carboplatin Shortages Underscore Need for Strategies to Prevent Future Cancer Drug Disruptions

Balazs Halmos, MD, explains why a national effort is needed to mitigate current and future drug shortages in oncology and discusses strategies being used to navigate the current cisplatin and carboplatin shortages.

New yorkUnited statesBalazs halmosDepartment of oncologyClinical cancer genomics at montefiore medical centerAlbert einstein collegeThoracic oncologyClinical cancer genomicsMontefiore medical centerShortage of cisplatin and carboplatinPatients with cancerAlbert einstein college of medicine

Study: Lowering the activity of KRAS gene can help fight pancreatic cancer

Study: Lowering the activity of KRAS gene can help fight pancreatic cancer If clinicians could stop mutations of the KRAS gene in pancreatic cancer – which happens in more than 90 percent of pancreatic cancer cases and drastically reduces response to immunotherapy – the chances of improving treatment for this deadly form of cancer would be increased. A collaborative study by Stony Brook University scientists, published in Nature Communications, takes an initial step toward better understanding how KRAS drives immune evasion and demonstrates a lowering of the KRAS activity resulting in a more favorable immune environment to fight cancer. Previous strategies to block the KRAS oncogene therapeutically have focused on counteracting its growth-promoting role in cancer.

Nancyc reichOleksi petrenkoEmily hendersonRenaissance school of medicineNature communicationsStony brook universityDepartment of microbiologyResearch assistant professorDepartment of pathologyStony brook university cancer centerRenaissance schoolStony brookScott powersClinical cancer genomicsStony brook university cancerPancreatic cancer