New research in worm models suggests that natural aging processes, as well as toxic beta-amyloid may drive the accumulation of harmful protein clumps associated with Alzheimer's disease. Boosting mitochondrial health may help delay the damage.
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Utilizing cutting-edge proteomics, researchers at the Buck Institute and elsewhere have mapped the "tau interactome" uncovering new findings about the role of tau in neurodegenerative disease. Publishing in Cell, scientists found that mutant tau impacts the function of mitochondria in human neurons.
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