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Scientists explore genetic origins of New Zealand s Kaimanawa wild horses

Samus Therapeutics Announces Study Publication in Communications Biology on the Importance of Epichaperome Inhibition for Enhancing the Vulnerability of Cancer Cells

Samus Therapeutics Announces Study Publication in Communications Biology on the Importance of Epichaperome Inhibition for Enhancing the Vulnerability of Cancer Cells

Samus Therapeutics Announces Publication of Study on Epichaperome Chemical Probes in Nature Communications

Samus Therapeutics Announces Publication of Study on Epichaperome Chemical Probes in Nature Communications
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Thirty-eight breeds tested for gene variant behind Warmblood fragile foal syndrome

Share The skeleton of the Arabian stallion Bairactar Or. Ar. (1813-1838) in the Stud Museum Offenhausen in Germany. A tooth from this skeleton was used for DNA analysis, and he was eliminated as the purported source of Warmblood fragile foal syndrome. Photo: Stephan Kube A study in which 4081 horses from 38 breeds were tested for the gene responsible for Warmblood fragile foal syndrome found an overall prevalence of 4.9%. Warmblood fragile foal syndrome is a recessive disorder, caused by a variant in the PLOD1 gene. Being recessive, it must be inherited from each parent for the condition to emerge in foals. Affected foals are either naturally aborted, stillborn, or euthanized. The distressing syndrome causes defects in connective tissue, meaning affected foals have hyper-extendible, abnormally thin, fragile skin that tears easily.

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