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Altering cellular interactions around amyloid plaques may offer novel Alzheimer's treatment strategies

Researchers made a significant breakthrough in Alzheimer s disease research by identifying a novel way to potentially slow down or even halt disease progression. The study, which focuses on the role of reactive astrocytes and the plexin-B1 protein in Alzheimer s pathophysiology, provides crucial insights into brain cell communication and opens the door to innovative treatment strategies.

Hongyan-zou
Roland-friedel
National-institute-on-aging
Medicines-partnership-alzheimer-disease-target-discovery
Associate-professor
Icahn-mount-sinai
Bin-zhang
Research-professor
Accelerating-medicines
Target-discovery
Preclinical-validation

New Alzheimer's Treatment Strategies May Alter Cell Interactions

New Alzheimer's Treatment Strategies May Alter Cell Interactions
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New-york
United-states
Hongyan-zou
Roland-friedel
Medicines-partnership
Icahn-school-of-medicine-at-mount-sinai
National-institute-on-aging
New-york-city
Icahn-school
Mount-sinai
Nature-neuroscience
Associate-professor

New Protein Target Could Halt Alzheimer's Progression

Researchers have found a new way to potentially slow or stop Alzheimer's disease. Their study shows that the plexin-B1 protein and reactive astrocytes (a

Roland-friedel
Hongyan-zou
Medicines-partnership
Icahn-school-of-medicine-at-mount-sinai
National-institute-on-aging
Icahn-school
Mount-sinai
Nature-neuroscience
Associate-professor
Icahn-mount-sinai
Bin-zhang
Research-professor

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