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 E-Mail Researchers have pinpointed a helper T cell population in the lungs of patients with severe COVID-19 that may be central to the development of hyperinflammation, lung injury, and subsequent acute respiratory distress syndrome (ARDS) during disease. Their data support the ongoing investigation of anti-cytokine therapies that target this cell subset, called tissue-resident memory-like Th17 cells (Trm17). To date, the bulk of research on immune responses to COVID-19 has mainly focused on T cells in the blood, while the role of tissue-specific immune cells in the inflamed lung has remained unclear. Accumulating evidence suggests that one of the causes of ARDS and, ultimately, death in COVID-19 patients is a hyperactive immune system, fueling exploration into drugs to block molecules that contribute to hyperactivation. Now, using a combination of single-cell RNA sequencing, cell surface protein sequencing, and T cell analyses, Yu Zhao and colleagues identified a helper T cell su

Yu zhaoMedicine healthImmunology allergies asthmaInfectious emerging diseasesயூ ழோமருந்து ஆரோக்கியம்நோயெதிர்ப்பு ஒவ்வாமை ஆஸ்துமாதடுப்பு மருந்துகள்தொற்று வளர்ந்து வருகிறது நோய்கள்