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New technology offers a faster, easier and more efficient way to study gene function

New technology offers a faster, easier and more efficient way to study gene function
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Hermana dierickKoenjt venkenNick matinyanEmily hendersonMarrs mclean department of biochemistryDanl duncan comprehensive cancer centerJohns hopkins university school of medicineBaylor college of medicineBaylor collegeCell reportstheJohns hopkins university schoolMarrs mclean departmentMolecular biologyஎமிலி ஹென்டர்சன்மார்ஸ் மக்லீந் துறை ஆஃப் உயிர் வேதியியல்ஜான்ஸ் ஹாப்கின்ஸ் பல்கலைக்கழகம் பள்ளி ஆஃப் மருந்து

Enzyme from fungi shows molecules which way to turn

Enzyme from fungi shows molecules which way to turn
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Tong zhuUnited statesRice universityYang gaoBaylor college of medicineZhiwen liuGeorge phillipsFanglong zhaoBoyang zhaoXue sherry gaoJoseph ferraraBiki bapi kunduBv venkataram prasadBanumathi sankaranNature communicationsRoberta welch foundation

Scientists investigate signal necrotic cells that cue phagocytic cells to engulf the dying cell

Scientists investigate signal necrotic cells that cue phagocytic cells to engulf the dying cell Just as people keep their houses clean and clutter under control, a crew of cells in the body is in charge of clearing the waste the body generates, including dying cells. The housekeeping cells remove unwanted material by a process called phagocytosis, which literally means eating cells. The housekeepers engulf and ingest the dying cells and break them down to effectively eliminate them. Phagocytosis is very important for the body s health, said Dr. Zheng Zhou, whose lab at Baylor College of Medicine has been studying phagocytosis for many years and provided key new insights into this essential process.

Yoshitaka furutaEmily hendersonMarrs mclean department of biochemistryBaylor developmentBaylor college of medicineTherapeutics graduate programBaylor danl duncan comprehensive cancer centerBaylor collegeMarrs mclean departmentMolecular biologyDisease modelsTherapeutics graduateDanl duncan comprehensive cancerஎமிலி ஹென்டர்சன்மார்ஸ் மக்லீந் துறை ஆஃப் உயிர் வேதியியல்பேலர் வளர்ச்சி

In a cell-eat-cell world calcium ions activate eat-me signal in necrotic cells

E-Mail Just as people keep their houses clean and clutter under control, a crew of cells in the body is in charge of clearing the waste the body generates, including dying cells. The housekeeping cells remove unwanted material by a process called phagocytosis, which literally means eating cells. The housekeepers engulf and ingest the dying cells and break them down to effectively eliminate them. Phagocytosis is very important for the body s health, said Dr. Zheng Zhou, whose lab at Baylor College of Medicine has been studying phagocytosis for many years and provided key new insights into this essential process. When this cell-eat-cell process fails, the dying cells will lose their integrity, break down and release their content into the surrounding tissues. Dumping the cell content would cause direct tissue damage and trigger inflammatory and autoimmune responses. If the dying cells are infected by a virus, releasing the cellular content would spread the infection.

Yoshitaka furutaOmar pena ramosMarrs mclean department of biochemistryBaylor developmentBaylor college of medicineLucia chiao at baylor college of medicineTherapeutics graduate programBaylor danl duncan comprehensive cancer centerNihondaihyou programBaylor collegeMarrs mclean departmentMolecular biologyDisease modelsTherapeutics graduateDanl duncan comprehensive cancerLucia chiao

Targeting RNA splicing can activate antiviral immune pathways in triple negative breast cancers

Targeting RNA splicing can activate antiviral immune pathways in triple negative breast cancers Researchers at Baylor College of Medicine have discovered how therapeutics targeting RNA splicing can activate antiviral immune pathways in triple negative breast cancers (TNBC) to trigger tumor cell death and signal the body s immune response. A new study published in Cell shows that endogenous mis-spliced RNA in tumor cells mimics an RNA virus, leading tumor cells to self-destruct as if fighting an infection. Researchers suggest this mechanism could open new avenues for turning on the immune system in aggressive cancers like TNBC. We know therapeutics that partially interfere with RNA splicing can have a very strong impact on tumor growth and progression, but the mechanisms of tumor killing are largely unknown. In this study, we discovered that these therapeutics are modulators of anti-tumor immunity, said Dr. Trey Westbrook, corresponding author of the study, executive director o

Jarey wangTrey westbrookElizabeth bowlingEmily hendersonTherapeutic innovation centerMarrs mclean department of biochemistryStudy coMcnair scholar in cancer research at baylorDepartment of molecularBaylor medical scientist training programResearchers at baylor college of medicineBaylor collegeMcnair scholarCancer researchStudy co first authorBaylor medical scientist training